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1. limit control and process 2. prevent and limit infection/further damage 3. Initiate adaptive immune response 4. initiate healing |
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Emigration of cells through the endothelial junctions |
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adherence of leukocytes to endothelial cells |
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thick clotted indicates more advanced inflammation |
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-manifestation of inflammation -increased numbers of circulating leukocytres |
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reaction of your own system WBC |
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borrow immune agents form another person ab |
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deleterious effect of hypersensitivity to environmental Ag |
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disturbance in the immunologic tolerance of self antigens |
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disturbance in the immunologic tolerance of self antigens |
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immune reaction to tissues of another individual |
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Hypersensitivity Type III |
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-cell mediated -no Ab -Tc, Th1 -graft rejection, Tb skin test, allergic rxns |
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haptens become immunogenic when they ind to host |
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What are the factors of infection |
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1.Mechanism of action 2.Infectivity 3. Pathogenicity 4. Virulence 5.Immunogenicity 6.Toxigenicity |
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Induction of long-lasting protective immune responses that will not result in disease in a healthy recipient |
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most common smallest works with complement to lyse cell |
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-found in blood stream -1st produced after 1st exposure (like after immunization) |
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-found in tiny amounts -stimulates B cells to divide and secrete Ab |
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produced by plasma cells under the skin -spit, tears, mucus, breast milk, saliva |
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-allergic reactions -found on basophils and mast cells |
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1.Precipitation 2. Neutralization 3. Opsonization 4. Complement activation |
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blood protein IgG to lyse cell walls |
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small polypeptides that cause vasodilatation causes swelling and produces pain |
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