Term
What is Prilosec's method of action? |
|
Definition
It's an antisecretory agent. |
|
|
Term
This med prevents post herpatic neuralgia... |
|
Definition
|
|
Term
|
Definition
Think of it as the opposite of insulin... it stimulates the release of blood sugar by the liver. Upon its secretion from the pancreas' alpha cells, it causes the liver to convert stored glycogen into glucose. |
|
|
Term
|
Definition
Rapid acting insulin. 15 minute onset. 60-90 minute peak. 3-4 hour duration. |
|
|
Term
|
Definition
Rapid acting insulin. 15 minute onset. 60-90 minute peak. 3-4 hour duration. |
|
|
Term
|
Definition
Only IV insulin. Short acting. 1/2-1 hour onset. Peak at 2-3 hours. Duration of 3-6 hours. |
|
|
Term
|
Definition
Cloudy insulin. Intermediate acting. onset in 204 hours, peak at 4-10 hours. Duration 10-16 hours. |
|
|
Term
|
Definition
Long acting insulin. Onset 1-2 hours. No peak. Duration 24+ hours. |
|
|
Term
|
Definition
Long acting insulin. Onset 1-2 hours. No peak. Duration 24+ hours. |
|
|
Term
|
Definition
Diuretic. Inhibits NaCl reabsorption. Decreases SVR and ECF. S/E: hypokalemia. Combined with triamterene for lower risk of hypokalemia. |
|
|
Term
|
Definition
Diuretic. Reduce K+ and Na+ exchange in distal collecting tubules. S/E hyper kalemia. Combined with hydrochlorothiazide to help overcome it's risk of hypokalemia. |
|
|
Term
|
Definition
Diuretic. Inhibits NA+ retaining and K+ excreting effects of aldosterone. S/E hyperkalemia, ED and lowered libido. |
|
|
Term
|
Definition
Centrally acting sympatholytic (alpha adrenergic antagonist). Reduces sympathetic outflow from CNS, reduces peripheral sympathetic tone, vasodilates, decreases systemic vascular resistance. S/E: dry mouth, ED, nausea/vomiting, sleep disturbance. Sudden d/c can cause withdrawal syndrome and rebound hypertenstion. |
|
|
Term
|
Definition
Alpha adrenergic blocker. Peripheral vasodilator. S/E: profound orthostatic hypotension after initial dose. Can also reduce resistance to outflow of urine in BPH. takind drug at bedtime reduces risks associated with orthostatic hypotension. Has a beneficial effect on lipid profile. |
|
|
Term
|
Definition
beta adrenergic blocker. antagonize beta adrenergic effects. decrease CO and reduces sympathetic vascular tone. Decreases renin secretion by kidneys. |
|
|
Term
|
Definition
beta adrenergic blocker. antagonize beta adrenergic effects. decrease CO and reduces sympathetic vascular tone. Decreases renin secretion by kidneys. |
|
|
Term
|
Definition
Direct vasodilator. reduces SVR and BP by direct arterial vasodilation. S/E: headache, nausea, flushing, tachycardia. IV used for htn crisis in hospital. not used as monotherapy because of S/E. contraindicated with CAD and being over 40 yoa. |
|
|
Term
|
Definition
ACE inhibitor. Inhibits angiotensin-converting enzyme. S/E: hypotension, hyperkalemia, acute renal failure. Aspirin and NSAIDs may reduce effectiveness. Don't use with potassium sparing diuretics. |
|
|
Term
|
Definition
Angiotensin II receptor blocker. Prevents the action of angiotensin II and produces vasodilation and increased salt and water excretion. S/E: hyperkalemia, decreased renal function. May take 3-6 weeks for full on effect on BP. |
|
|
Term
|
Definition
calcium channel blocker. blocks movement of intracellular calcium into cells. causing vasodilation and decreased HR, contractility, and SVR. S/E: bradycardia, 1st degree heart block, nausea, headache, dizziness, peripheral edema, flushing, rash. Use with caution in patients with heart failure. Avoid grapefruit! |
|
|