Term
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Definition
| periodic change in the antigen displayed by the pathogen to avoid immune response. |
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Term
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Definition
| proteins used to induce phagocytosis of pathogens |
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Term
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Definition
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Term
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Definition
| attachment of pathogen to host by adhesins or ligands that bind to complementary surface receptors on host tissues |
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Term
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Definition
| bacterial enzymes that clot the fibrinogen in the blood |
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Term
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Definition
| Bacterial enzymes that break down fibrin and digest clots formed by the body to isolate the infection. |
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Term
| Hemolytic Patterns of Kinases (Alpha) |
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Definition
| Partial pattern with green tint |
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Term
| Hemolytic Patterns of Kinases (Beta) |
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Definition
| Complete pattern with yellow tint |
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Term
| Hemolytic Patterns of Kinases (Gamma) |
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Definition
| No defined pattern with no tint |
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Term
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Definition
| enzyme secreted by certain bacteria to hydrolyze hyaluronic acid to break apart the cells in particularly the connective tissue cells. |
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Term
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Definition
| An enzyme produced by many Clostridium species that break down the protein collagen. |
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Term
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Definition
| enzymes that destroy IgA antibodies |
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Term
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Definition
| proteins that are poisonous substances produced by certain microorganisms |
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Term
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Definition
| produced inside some bactera as part of their growth and metabolism and are secreted by the bacterium into the surrounding medium or release following lysis |
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Term
| A-B toxins (exotoxin type) |
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Definition
the exotoxin consisting of an active enzyme component (A) and a binding component (B).
(i.e. Diphtheria) |
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Term
| Membrane Disrupting Toxin |
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Definition
| cause lysis of the host cells by disrupting their plasma membranes. |
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Term
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Definition
| antigens that provoke a very intense immune response by causing T cells to release cytokines that enter the bloodstram and give rise to many S/S (fever, nausea, vomiting, diarrhea, shock and sometimes death) Examples: food poisoning staphylococcal toxins and TSS |
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Term
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Definition
| part of the outer portion of the cell wall of gram-negative bacteria. The outer wasll consists of lipoproteins, phospholipids, and lipopolysaccharides (LPS). Lipid A is an LPS endotoxin.Endotoxins are activated when the Gram neg bacteria lyse. S/S include chills, fever, weakness, generalized aches, sometimes shock or death. Endotoxins may induce a miscarriage. |
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Term
| Mircobial Mechanisms of Pathogenicity |
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Definition
1. Portal of Entry
2. Invasion of Microbes/Adherence
3.Penetration or Evasion of Host Defenses
4. Damage to Host Cells
5. Portal of Exit (usually same route as entry) |
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