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Epidermis Dermis Subcutaneous (adipose or fat) |
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Protein produced by keratinocytes, making the epidermis waterproof |
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The white, crescent shaped portion of the nail at the lower end of the nail plate |
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Produced by sebaceous glands. Mildly bacteriostatic substance that lubricates the skin and reduces water loss from surface. |
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Protection Homeostasis (water balance) Temperature regulation Sensory organ Vitamin D synthesis Psychosocial |
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Initial reaction to a problem that alters one of the structural components of the skin |
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Changes in the appearance of a primary lesion |
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Thickening of skin, can come from continual rubbing of area due to itching |
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Asymmetry of shape Border irregularity Color variation within one lesion Diameter greater than 6mm Evolving or change in any feature |
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Macules, nodules, patches, cysts, papules, vesicles, bullae, plaques, pustules, wheals, erosions |
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Primary Lesion Flat lesions less than 1cm in diameter. Color different from surrounding skin. |
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Elevated marble like lesions more than 1cm wide and deep |
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Macules that are larger than 1cm in diameter. May or may not have some surface changes (slight scale or fine wrinkles) |
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Nodules filled with either liquid or semisolid material that can be expressed |
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Small, firm, elevated lesions less than 1cm in diameter |
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Blisters filled with clear fluid. Less than 1cm in diameter |
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Blisters filled with clear fluid. More than 1cm in diameter. |
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Elevated, plateau like patches more than 1 cm in diameter that do not extend into the lower skin layers. |
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Vesicles filled with cloudy or purulent fluid |
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Elevated, irregularly shaped, transient areas of dermal edema |
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Wider than fissures, but only involve the epidermis. Often associated with vesicles, bullae, or pustules |
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Scales, ulcers, crusts and oozing, lichenifications, fissures, atrophy |
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Visibly thickened stratum corneum. Appear dry and are usually whitish. Most often seen with plaques and pustules |
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Deep erosions that extend beneath the epidermis and involve the dermis and sometimes subcutaneous fat |
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Composed of dried serum or pus on the surface of skin, under liquid debris may accumulate. From broken vesicles, bullae, or pustules |
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Palpably thickened areas of epidermis with accentuated skin markings. Usually caused by chronic rubbing and scratching |
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Linear cracks in epidermis which often extend into dermis |
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Characterized by thinning of the skin surface with loss of skin markings. Skin is translucent and paper like |
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Small, reddish purple lesions that do not fade or blanch when pressure is applied |
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Excessive growth of body hair or hair growth in abnormal body areas |
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Inflammation of the skin around the nail |
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Inflammation of the skin around the nail that persists for months |
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Healing in which the wound can be easily closed and dead space eliminated without granulation, which thus shortens the phases of tissue repair. Results in thin scar |
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The formation of scar tissue for wound healing to occur |
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Healing of deep tissues or wounds with tissue loss in which a cavity-like defect requires gradual filling of the dead space with connective tissue. Prolongs the wound healing process |
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Delayed primary closure of a wound with a high risk for infection. Wound is intentionally left open for several days until inflammation has subsided and then is closed by first intention |
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Skin is intact. Area is red and does not blanch |
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Skin is not intact. There is a partial-thickness skin loss of the epidermis or dermis. |
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Skin loss is full thickness. Subcutaneous tissues may be damaged or necrotic. Does NOT extend to muscle or bone. |
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Skin loss is full thickness with exposed or palpable muscle, tendon, or bone. |
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Black, grey, or brown nonviable, denatured collagen |
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Separation of the skin layers at the wound margins from the underlying granulation tissue |
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Superficial infection involving the hair follicle |
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Severe pain persisting after Shingles has resolved |
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Infestation by human lice (head or body) |
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Contagious skin disease caused by mite infestations |
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Cysts, seborrheic keratoses, keloids, nevi |
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Firm, flesh-colored nodules that contain liquid or semi solid material |
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An overgrowth of a scar with excessive accumulations of collagen and ground substance |
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Squamous cell carcinoma, basal cell carcinoma, melanoma |
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Cancer of the epidermis. Potentially metastatic. Chronic skin damage from repeated injury or irritation can predispose |
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Arise from basal cell layer of epidermis. Metastasis is rare. UV exposure most common cause. |
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Pigmented cancers arising from melanin-producing cells. Highly metastatic. |
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Whiteheads and blackheads |
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Often drug-induced immunologic skin reaction |
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Abraded area where the skin is town or torn off. Can be due to scratching, funhal infection, incontinence, etc |
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Caused by reactivated Varicella-zoster virus. S/S: burning/sharp/stabbing pain/aching/itching. Linear pattern of vesicles along a dermatome. |
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Treatment of Herpes Zoster |
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Antiviral agents Systemic corticosteroids Analgesics Neuroleptics |
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Systemic factors that influence skin |
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General health and functioning Incontinence Nutrition Blood sugar/diabetes Infection Medications Stress Smoking |
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What to address to optimize wound healing |
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Address etiology (most important) Systemic factors (circulatoin, oxygenation, nutrition, diabetic management) Local factors (infection, perfusion, edema, incontinence, pressure relief) Topical therapy (last to be addressed) |
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Remove necrotic tissue Maintain clean, moist wound surface Identify and treat infection Fill dead space Absorb exudate Insulate wound surface |
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Screening tool designed to determine pts risk of developing a pressure ulcer 6 subscales add together for a value from 6-23 Lower the score, higher risk of developing breakdown |
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What to assess when you find a pressure ulcer |
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Location on body Size: length, width, depth, undermining, tunneling Stage: I-IV Wound bed type Drainage: type, amt Peri-wound skin: color, condition |
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Tissue types in wound bed |
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Epithelialization: pink and dry Granulation: red and moist Slough: yellow Eschar: black |
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