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mainly exists in one plce: trauma, tumors, inflammatory, and vascular. NOT SEEGMTNAL DISRODER- lONG TACT |
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the tracts, secondaryto segmental. Primary longitudinal rpoblems: heritbable metabolic,ysstemic disease. |
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dissociated snesiory loss (on one space), dsorsal column vs. spinothalamic ; sacral sparig (?) |
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painful, peripheal nerves |
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cns (brain/spinal cord) are painless |
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abronmal sensatino from normal stimulus |
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normal stimulus leadign to uncofmortable sensation |
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loss of position sensation? |
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nocturia, urinary frequency, bowel/bladderi nconcinentc . need to get bilater loss for this to happen. Horners syndrome- miosis, |
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two dermatomes ahve to be hit to lose sensation peripeharlly |
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look at slide of spinalw levels |
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see that fassiculus cuneatus doesntstart until cervial levels and is on outside ofr FG. see that graciullus starts on sacral levels nad ontiues to get bigger sa you rise up. |
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lsitent o superior cerviacl ganglion slide |
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Hypothalathalamic system goes throug here goes down and synapses in T1,t2. any disruptions i nsuperior c ervical gna glion or t1,t2 will lead to horners syndrome. Goes down laterally |
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everythign bloew lesion si a mess- two sensory pathways, and are disconnected. Cut othe only descending motor pathway bilaterally. UMN lesion. LMNs are there, but are not being controleld by your head. all the uMN symptoms with noen of the lMNS |
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complete transection causes |
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fracture rauma, bullet/knife, demyelianting disease (MS), compression by tumor/inflammatory mass. Also lose bladder conrol |
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hemiseciton0 same side coritcospinal adn dorsal column, spinaltholamic are contralateral. but also lose LMN funcitno at that specific level (but not below). AUtonomics- horners syndrome (misos,ptosis, anhydrosis) |
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extramedullary compression |
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look up. dorsal root compression- pain and parasthesia follwoed by los of sensation. Reflex loss due ot lost sneosry input. Motor loss of ipsilateral motor roots (LMN), and ipsilateral uMN syndrome below . |
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disc herniation lcoations |
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medial disc herniation will cause compresison fo multiple levels of cauda equina, while lateral will cause just one compression |
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spyngomyelafluid filled cavity in center of the cord. first presents with s egmental snseosry adn motor loss, then spreads to motor ascending/descendign pathway symtpoms. initial symptoms are loss of pain and etmperaure across shoudlers, nd lateral arms. in teh enD presents iwth loss of pain/temp,motor neruons loss all over except there is sacral sparing |
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ALS- amytrophic lateral sclerosis |
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atrophci weakness of hands/forearms, spaciticy of legs- hyperreflexia: a mixed uMn/LMN dissorder, progressive spread rostrally and caudally until fatal. only motor neurons efected, no snesory involvement |
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subacute combined defieicny.macrocytic anemia . DOrsal columns are most affected, and croticospian trac tenxt. Symmetric snesory loss. B12 deficiency causes periphral neuropaty. paradoxial cobo of extensor plantar refelx (UMN sin) and hypoactive ankle |
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dissemianted lesiosn in temime and space. Dorsal column, CST, cerebellar |
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anteior spinal artery losss effect |
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loss of pain ant empt bilat, loss cortiscopinal tract, but dorsal colum will be preserved. Most omcmonly cocurs at arter of admkiewice (t12-l2) |
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affecst entire cross sectional area of spinal cord |
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most affects are caused yb comrpesison by teh AVM- readicualr pain leag weakness, bladder distrubacne |
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severe radicualr pain, urinary /rectal icnontinence, muscle atrophy and arrefelxia in leg (unilateral) |
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severe radicualr pain, urinary /rectal icnontinence, muscle atrophy and arrefelxia in leg (unilateral) |
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severe radicualr pain, urinary /rectal icnontinence, muscle atrophy and arrefelxia in leg (unilateral) |
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bilateral pain, bilateral sensory loss, (reflexes itnact), sever incotniennace |
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