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refers to the how good the heart contracts |
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work imposed on the heart prior to contraction |
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work with healthy muscle fibers. It measures the contractility of the cardiac muscles |
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Tension that develops in ventricular wall during systole |
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Volume Systemic vascular resistance ventricular dilation increased volume |
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uses calcium. Ability of muscle fibers to shorten during contraction |
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what affects contractility |
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myocardial oxygen consumption CAD Cardiac muscle disease Ca+ or K+ imbalances |
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decrease ventricular filling time - possible decrease stroke volume & decrease CO. Works with different tachy rythms |
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longer ventricular filling time & possible increase stroke volume & increase CO |
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where the baroreceptors are |
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carotid sinus and aortic arch |
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unresponsive baroreceptors |
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bellow 50 mm Hg or above 200mm Hg. |
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manipulate the carotid in tachy pts |
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renin. produces angeiotensin - produces aldosterone |
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atrial natriuretic factor from atrial tissue |
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released in response of increased B/P |
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5 signs&symptoms of heart disease |
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Dyspnea Chest pain Syncope Palpitations Edema |
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on exertion positional - sign of HF paroxymal nocturnal - sign of L ventricular failure orthopnea - sign of HF |
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Intermittent, severe, sharp Sudden onset Under or to left of sternum Worsened with: Inspiration Swallowing Coughing Turning upper body Relieved by sitting up & leaning forward |
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Transient loss of consciousness caused by cerebral hypoxia |
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Heart block or other rhythm disturbances Sudden, unexpected loss of consciousness May be loss of pulse for 15-30 seconds |
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Hypersensitive carotid sinus syncope |
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Vagal syncope Most common in older men with atherosclerotic carotid arteries Fainting precipitated by: Sudden turning of the head Shaving neck Tight collar Inadvertent pressure to carotid artery sinus below jaw angle |
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Sensation of rapid “racing” heartbeat Sensation of skipped heartbeats |
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Abnormal accumulation of serous fluid in soft tissues |
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Sensation of rapid “racing” heartbeat Sensation of skipped heartbeats |
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Sensation of rapid “racing” heartbeat Sensation of skipped heartbeats |
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R bundle branch block Pulmonary hypertension R ventricular failure from septal defect. R sided cardiac issues |
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Indicates rapid filling of ventricles Normal in children & up to age 25 – 30 After age 30 – called a “gallop” Usually related to ventricular failure |
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atrial diastolic gallop Produced by high velocity blood flow during atrial contraction as blood enters a noncompliant ventricle. turbulent flow of blood |
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maximum amount of oxygen your heart can provide to your muscles during sustained activity |
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HR and rhythm during normal patient activities |
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Invasive Hemodynamic Monitoring |
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Swan-Ganz Catheter Central venous pressure Pressure in R atrium 2 – 8 mm Hg Pulmonary artery pressure Indirect measurement of L ventricle pressure 8 – 10 mm Hg |
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To treat heart failure Strengthens contractions up SV & CO blood to kidneys and up urine output down blood volume & down preload Digoxin (Lanoxin) |
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Vasoconstriction Bronchial Dilation |
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Very Nice Drugs” Verapamil Nifedipine Diltiazem |
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rapid precipitous drop in B/P |
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used in emergencies & ICU’s only sympathomimetics Increase CO & B/P Dopamine Dobutamine |
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Class I Local anesthetic effect Ventricular Dsyrhythmias Class II Beta Adrenergic Blockers – “olols” Slow cell recovery and automaticity Class III Block potassium channels Slows rate and conduction Class IV Calcium Channel Blockers |
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Class I: Sodium Channel Blockers |
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Lidocaine - prototype Stabilize cell membrane anesthetic effect Caution Heart block 3rd degree block CHF Hypotension Shock Renal/hepatic disease Quinidine, procainamide |
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Propranolol – prototype Antihypertensive, anti-anginal, antimigraine SVT’s from digoxin or catecholamines |
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Class III: K+ Channel Blockers |
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Sotalol (Betapace) – Prototype Documented life-threatening rhythms Maintain NSR after conversion of A-fib Amiodarone (Cordarone) Documented life-threatening rhythms Potentially fatal liver toxicity Bretylium V- fibrillation Not responding to other drugs |
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Class IV: Calcium Channel Blockers |
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Diltiazem (Cardizem) – prototype Paroxysmal SVT A-fib A-flutter Verapamil |
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Intra-aortic balloon counterpulsation |
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Cardiogenic shock Following massive MI Following surgery Supports CO |
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– To treat heart failure – Strengthens contractions SV & CO blood to kidneys and urine output blood volume & preload Digoxin (Lanoxin) |
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• ACE Inhibitors – the “prils” |
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– the “olols” – Non-specific – Propranolol (Inderal) – Nadolol (Corgard) |
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– Specific; bronchial effects – Patenolol (Tenormin) – Metoprolol (Lopressor) |
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– Vasodilation – Bronchial Dilation |
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• Calcium Channel Blockers |
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Very Nice Drugs” • Verapamil • Nifedipine • Diltiazem |
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• Angiotensin II Receptor Blockers – the “tans" (ARBs) |
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– Losartan (Cozaar) – Candesartan (Atacand)\ • Prevent vasoconstriction • Prevent aldosterone release |
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• Act on smooth muscle walls of blood vessels to widen lumen and resistance increased flow |
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used in emergencies & ICU’s only – sympathomimetics – Increase CO & B/P • Dopamine • Dobutamine |
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decrease venous return to the heart |
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Atrial Junctional Heart Block Ventricular |
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#1 priority in the tPA treatment |
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CHF compensatory mechanisms |
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Frank-Starling mechanism Increase HR Ventricular hypertrophy Ventr. dilation Baroreceptor stimulation |
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Dyspnea – all types Exertional, at rest, orthopnea & paroxysmal nocternal dypnea Dry, nonproductive cough *Moist crackles on auscultation |
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relate to systemic back-up of blood Distended neck veins Peripheral edema Pitting edema Worsened by Aldosterone & ADH Venous congestion Hepatomegaly |
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Pleural Effusion Dysrhythmias Left Ventricular Thrombus Hepatomegaly Renal failure |
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which heart block progresses the least into 3rd degree |
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Thoracic and abdominal aneurysms may be asymptomatic. Back or abdominal pain Bruit Edema of UE, s/s of embolization On physical exam, the patient may have a pulsatile abdominal mass. DO NOT PALPATE!! |
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6 P's of arterial obstruction |
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Pain Pallor Pulselessness Paresthesias Paralysis Poikilothermia |
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Edema Pain Warmth Erythema Elevated temperature Homan’s sign |
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