Term
1. The 3 layers of arteries are the _____, _____ and the _____. |
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Definition
TUNICA INTIMA, TUNICA MEDIA, TUNICA ADVENTITIA |
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Term
2. The _____ layer of arteries is where lesions and fatty deposits mostly occur. |
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Definition
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Term
3. 3 components of the tunica intima layer of arteries. |
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Definition
1) ENDOTHELIUM, 2) SUBENDOTHELIUM, 3) ELASTIC LAYER |
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Term
4. The _____ layer of blood vessels is partially where lesions occur. |
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Definition
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Term
5. 2 components of the tunica media layer of arteries. |
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Definition
1) SMOOTH MUSCLE, 2) ELASTIC FIBERS |
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Term
6. What makes up the tunica adventitia layer of arteries? |
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Definition
LOOSE CONNECTIVE TISSUE WITH NOURSIHING VESSELS (VASA VASORUM) |
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Term
7. 6 components of the recurrent injury hypothesis of atherosclerosis. |
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Definition
1) RECURRENT INJURY, 2) ENDOTHELIAL DISRUPTION, 3) MONOCYTE ADHERENCE AND ACTIVATION, 4) SMOOTH MUSCLE CELL PROLIFERATION, 5) ACCUMULATION OF LIPIDS, 6) ORGANIZATION OF MATURE PLAQUE |
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Term
8. _____ can occur beneath the endothelium of arteries, and can easily disrupt and cause almost immediate clotting. |
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Definition
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Term
9. 2 alternatives to statin drugs are _____ and _____ which can help with cardio health. |
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Definition
CHINESE RED RICE EXTRACT, POLYCOSANOLS |
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Term
10. Because lipids are insoluble in plasma, they are encapsulated by certain fat-carrying proteins called _____. |
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Definition
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Term
11. 5 different classes of lipoproteins. |
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Definition
1) CHYLOMICRONS, 2) VLDL’S, 3) IDL’S. 4) LDL’S, 5) HDL’S |
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Term
12. _____ are lipoproteins synthesized by the small intestine and liver. They are involved in the transport of exogenous triglycerides and cholesterol that has been absorbed from the GI tract. |
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Definition
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Term
13. _____ are lipoproteins synthesized by the liver. They are the primarily endogenous pathway for transform of triglycerides that are already in the body. |
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Definition
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Term
14. VLDL’s carry triglycerides to tissue capillaries for entry into _____ and _____ cells. |
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Definition
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Term
15. _____ constitute the lipoprotein remnants that remain when the triglycerides are removed from the VLDL. |
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Definition
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Term
16. IDL’s are either taken up and broken down by the _____, or they are converted to _____. |
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Definition
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Term
17. _____ are the major carriers of cholesterol, so its measurements provide good estimates of blood cholesterol. |
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Definition
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Term
18. About 2/3 of the circulating LDL’s are removed by the _____ by means of the LDL receptors. The remainder is taken up and broken down in peripheral tissues by degradative process which includes _____. |
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Definition
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Term
19. When macrophages uptake LDL within the arterial wall, it can result in these 3 things. |
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Definition
1) ACCUMULATION OF ESTER, 2) FORMATION OF FOAM CELLS, 3) DEVELOPMENT OF ATHEROSCLEROSIS |
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Term
20. _____ serve as the carriers that remove cholesterol from the peripheral tissue, and transport it back to the liver for catabolism and excretion. They also inhibit cellular uptake of LDL’s. |
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Definition
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Term
21. High levels of _____ contribute to the development of artherosclerosis, whereas high levels of _____ appear to provide some protection against the disorder. |
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Definition
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Term
22. 3 groups of people who have elevated levels of HDL’s. |
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Definition
1) PEOPLE WHO EXERCISE REGULARLY, 2) WOMEN WHO TAKE ESTROGENS, 3) PEOPLE WHO CONSUME MODERATE AMOUNTS OF ALCOHOL |
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Term
23. _____ and _____ are associated with high levels of LDL’s and low levels of HDL’s. |
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Definition
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Term
24. LDL’s are the major carriers of _____, and form _____. |
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Definition
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Term
25. VLDL’s are the major carriers of _____. |
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Definition
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Term
26. HDL’s are the good guys in that they prevent _____ formation. |
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Definition
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Term
28. 5 major risk factors of cardiovascular disease that can be changed. |
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Definition
1) CIGARETTE SMOKING, 2) HIGH BP, 3) BLOOD CHOLESTEROL LEVELS, 4) DIABETES, 5) STRESS |
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Term
29. 4 major risk factors of cardiovascular disease that cannot be changed. |
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Definition
1) HEREDITY, 2) SEX, 3) RACE, 4) AGE |
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Term
30. _____ is the core/root of angina. |
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Definition
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Term
31. This type of ischemia does not have symptoms or angina. |
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Definition
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Term
32. If a person presents with ischemic hypoxia, their ECG will show _____ and might have a _____. |
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Definition
ST SEGMENT DEPRESSION, T WAVE INVERSION |
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Term
34. In this type of hypoxia, there is a lack of O2 to cells due to a lack of oxygen loading in the blood. |
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Definition
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Term
35. In this type of hypoxia there is a lack of blood flow due to restricted O2 causing cellular swelling. |
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Definition
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Term
36. If a person has ischemic hypoxia, their cells will lose the ability to do these 2 things. |
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Definition
1) LOSE THE BAILITY TO SYNTHESIZE PROTEINS, 2) LOSE CELL MEMBRANE PERMEABILITY |
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Term
37. This type of hypoxia is caused by a lack of O2 to cells due to decreased RBCs and/or hemoglobin. |
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Definition
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Term
38. This type of hypoxia is caused by a toxin that caused oxygen deprivation in the tissues. |
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Definition
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Term
39. _____ is the number one cause of cell injury. |
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Definition
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Term
40. When cell injury is sustained due to hypoxia, _____ keeps the platelets from getting sticky, and sometimes it removes the clot and restores the flow of blood in the lumen. |
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Definition
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Term
41. When is cell intervention too late/ what is the point of no return? |
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Definition
WHEN MITOCHONDRIA FILL WITH CALCIUM, WHICH DESTROYS ATP MAKERS |
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Term
42. O2 is necessary to manufacture _____ in cardiac cells. |
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Definition
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Term
43. When cardiac cells are in O2 deprivation, it goes anaerobic for a while and causes a build up of _____. |
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Definition
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Term
44. Eventually the lack of ATP will cause _____ failure. |
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Definition
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Term
45. When there is NA/K pump failure, sodium and water accumulate inside the cell causing _____. |
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Definition
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Term
46. _____ is an early sign of cell injury. |
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Definition
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Term
47. Cellular swelling is _____. |
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Definition
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Term
48. When cellular swelling occurs, organelles become distorted, disrupted, and porous affecting the cell membrane in these 2 ways. |
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Definition
1) RIBOSOMES CAN’T MANUFACTURE PROTEINS BECAUSE OF DISTORTION, 2) POROSITY OF MEMBRANE WILL ALLOW CALCIUM TO ACCUMULATE IN THE CELL WHICH IS A HALLMARK OF CELL INJURY, 3) MITOCHONDRIA BURST |
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Term
49. 4 signs and symptoms of hypoxia. |
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Definition
1) DIAPHORESIS, 2) ANXIETY, 3) GREAT PAIN, 4) NAUSEA |
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Term
50. If there is cellular injury occurring an ECG will show a _____ and _____. |
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Definition
ST ELEVATION AND WIDENED Q WAVE |
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Term
51. If cellular injury occurs, blood work will show up _____ hours after cell injury. |
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Definition
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Term
52. If cellular injury occurs the blood work will show an elevation of these 5 cardiac enzymes. |
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Definition
1) CK, 2) CPK, 3) CPK-MB, 4) SGOT/AST, 5) LDH |
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Term
53. _____ begins rising 3-4 hours after cellular injury. |
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Definition
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Term
54. Is found in the actin and myosin of muscle cells, and begins rising 4-6 hours after cellular injury. |
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Definition
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Term
55. _____ is responsible for O2 storage in muscle cells, and begins rising 1-2 hours after injury. |
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Definition
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Term
56. _____ begins rising 4-6 hours after cellular injury. |
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Definition
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Term
57. If there is cellular injury WBC count will be _____ within 12-24 hours. _____ is usually due to systemic infection with is part of the inflammatory response. |
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Definition
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Term
58. 4 manifestation categories of a myocardial infarction. |
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Definition
1) PAIN AND AUTONOMIC RESPONSES ASSOCIATED WITH ISCHEMIC EVENTS, 2) WEAKNESS AND SIGNS RELATED TO IMPAIRED MYOCARDIAL FUNCTION, 3) DYSRHYTHMIA, 4) ECG CHANGES |
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Term
59. 3 ECG changes that will occur in a patient with a MI. |
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Definition
1) ST ELEVATION, 2) T WAVE INVERSION, 3) WIDE Q WAVE |
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Term
60. The ECG changes that occur in a patient with an MI are associated with _____ and _____ of myocardial cells. |
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Definition
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Term
61. 7 complications of an MI. |
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Definition
1) SUDDEN DEATH, 2) HEART FAILURE, 3) PERICARDITIS, 4) THROMBOEMBOLI, 5) RUPTURE OF MYOCARDIUM, 6) ANEURYSM, 7) ARRHYTHMIAS |
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Term
62. Pain from myocardial infarctions present as _____ and _____ pain. |
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Definition
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Term
63. The vagal stimulation from myocardial infarction will cause _____ and _____. |
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Definition
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Term
64. The sympathetic stimulation from myocardial infarction will cause _____, _____ and a _____. |
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Definition
TACHYCARDIA, ANXIETY, AND AN INCREASE IN BP |
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Term
65. Following a myocardial infarction, no tissue changes are noted in the first _____ hours. |
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Definition
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Term
66. After _____ hours, the tissue turns pale to grey-brown, with slight pallor of the area. |
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Definition
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Term
67. After _____ days following a myocardial infraction, necrosis of the area is apparent In the center and hyperemic around the edges. |
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Definition
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Term
68. After _____ days following a myocardial infarction, the area becomes soft and fatty changes in the center are well developed. Hemorrhagic areas are present in the infarcted areas. |
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Definition
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Term
69. After _____ days following a myocardial infarction, fibrotic scar tissue replacement occurs and revascularization commences. |
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Definition
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Term
70. After _____ weeks following a myocardial infarction, scar tissue replacement of necrotic tissues ensues. |
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Definition
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Term
71. _____ is the number 1 killer of people in the US. It is a disease that manifests within the lumen of the coronary arteries, or beneath the endothelium. |
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Definition
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Term
72. _____ is the most common artery affected by coronary artery disease. |
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Definition
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Term
73. _____ is the major symptom of coronary artery disease. |
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Definition
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Term
74. 3 types of angina that present in coronary artery disease. |
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Definition
1) CLASSIC ANGINA, 2) UNSTABLE ANGINA, 3) VARIABLE ANGINA |
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Term
75. _____ is angina that is caused by an increased workload on the heart during physical stress. It often goes away after rest. |
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Definition
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Term
76. _____ is angina that is 1 step closer to cell injury and infarction. There are periods of some blocking where it cuts off the blood flow beyond the lesion. During this period of time there can be partial or total obstruction. |
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Definition
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Term
77. Unstable angina is not associated with exercise, rather it comes during _____. |
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Definition
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Term
78. _____ is a vasodilator used with unstable angina that is a vasodilator used to bring relief of symptoms by improving oxygen supply. |
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Definition
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Term
79. _____ is vasospastic angina not usually associated with atherosclerosis. Instead it is associated with spastic vessels. |
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Definition
VARIABLE ANGINA (PRINZMETAL’S ANGINA) |
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Term
80. _____ will be the final outcome of variable angina in the _____ arteries. |
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Definition
HYPOXIA; CORONARY ARTERIES |
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Term
81. _____ is fibrous banding that encases the heart like a cast. The heart can’t fill or pump very well, causing a life threatening situation. |
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Definition
CONSTRICTIVE PERICARDITIS |
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Term
82. 6 impaired cardiac functions that can cause heart failure. |
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Definition
1) CARDIOMYOPATHY, 2) MI, 3) CORONARY INSUFFICIENCY, 4) CONGENITAL DEFECTS, 5) CONSTRICTIVE PERICARDITIS, 6) VULVAR HEART DISEASE |
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Term
83. 3 excess work demands of the heart that can eventually lead to heart failure. |
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Definition
1) INCREASED PRESSURE CAUSING STRICTURING OF THE AORTA, 2) INCREASED VOLUME WORK, 3) INCREASED PERFUSION WORK |
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Term
84. _____ causes congestive blood back up into the lungs eventually leading to pulmonary edema. |
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Definition
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Term
85. If left sided heart failure is present in a patient, a doctor will hear _____ or _____, and the patient might also present with _____ because of collapsed alveoli. |
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Definition
RALES, CRACKES; ABSENT BREATH SOUNDS |
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Term
86. If a patient presents with left sided heart failure, the backup will go all the way back and stress the _____. |
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Definition
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Term
87. The number one cause of right sided heart failure is _____. |
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Definition
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Term
88. If left sided heart failure causes right sided heart failure, the venous blood will back up to the jugular veins and one will see _____. |
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Definition
JUGULAR VENOUS DISTENTION |
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Term
89. 7 signs and symptoms of a patient with left sided heart failure. |
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Definition
1) EXERTIONAL DYSPNEA, 2) ORTHOPNEA, 3) PAROXYSMAL NOCTURNAL DYSPNEA, 4) PRODUCTIVE COUGH, 5) BLOOD-TINGED SPUTUM, 6) CYANOSIS, 7) ELEVATION IN PULMONARY CAPILLARY WEDGE PRESSURE |
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Term
90. To test elevation in pulmonary capillary wedge pressure during left sided heart failure use a _____, which is threaded through the right ventricle, pulmonary trunk, and it measures the left ventricular pressure. |
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Definition
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Term
91. 3 treatments used for left sided heart failure that can also help right sided heart failure. |
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Definition
1) DIGOXIN, 2)ACE INHIBITOR, 3) BIODIN FOR AFRICAN AMERICANS |
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Term
92. 9 symptoms of right sided heart failure. |
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Definition
1) HEPATOMEGALY, 2) SPLENOMEGAY, 3) PORTAL HYPERTENSION, 4) FATIGUE, 5) DEPEDENT EDEMA, 6) JUGULAR VEIN DISTENSION, 7) ANOREXIA, 8) CYANOSIS, 9) PERIPHERAL VENOUS PRESSURE |
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Term
93. 3 bodily compensatory mechanisms when tissues are not getting enough O2. |
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Definition
1) SYMPATHETIC NERVOUS SYSTEM, 2) RENIN-ANGIOTENSIN-ALDOSTERONE, 3) ANAEROBIC METABOLISM |
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Term
94. 4 responses of the sympathetic nervous system in response to tissues not getting enough O2. |
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Definition
1) INCREASES HEART RATE, 2) INCREASED MYOCARDIAL CONTRACTILITY, 3) INCREASED WORK DEMAND ON THE HEART, 4) VASOCONSTRICTION LEADING TO INCREASED PVR |
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Term
95. 4 ways the renin-angiotensin-aldosterone system compensates in response to tissues not getting enough O2. |
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Definition
1) JGA CELLS DETECT A DECREASE IN BLOOD PRESSURE AND RELEASE RENIN, 2) RENIN IS CONVERTED TO ANGIOTENSINOGEN IN THE LIVER WHICH CONVERTS TO AG2, 3) THIS CAUSES THE BODY TO RETAIN MORE SODIUM AND WATER INCREASING BLOOD VOLUME, 4) THE INCREASED VOLUME INCREASES THE WORK LOAD ON THE HEART INCREASING PVR |
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Term
96. 3 steps in how anaerobic metabolism compensated in response to tissues not getting enough O2. |
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Definition
1) O2 NOT GETTING TO CELLS DECREASES ATP PRODUCTION, 2) THIS CAUSES A BUILD UP OF LACTIC ACID, 3) ACIDOSIS IMPAIRS CONTRACTILITY OF THE HEART |
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Term
97. 9 steps in cell injury |
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Definition
1) DECREASED O2 TO CELLS, 2) DECREASED ATP SYNTHESIS, 3) FAILURE OF THE NA/K PUMP, 4) SODIUM ACCUMULATES INSIDE THE CELL CAUSING WATER TO FLOW IN, 5) CELLULAR SWELLING INSUES, 6) THIS CAUSES CELLULAR DISTORTION DAMAGING ORGANELLES, 7) PROTEIN SYNTHESIS DECLINES, 8) CELL MEMBRANES BECOME PERMEABLE, 9) CALCIUM ENTERS THE CELL DESTROYING MITOCHONDRIA |
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Term
98. 8 noninvasive assessments of cardiovascular function. |
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Definition
1) SENSORIUM, 2) MUCOUS MEMBRANE COLOR, 3) PULSE RATE, 4) AUSCULTATION OF HEART SOUNDS, 5) CARDIOGRAPHY, 6) DOPPLER STUDIES, 7) STRESS TESTING, 8) CHEST X-RAYS |
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Term
99. 3 invasive assessments of cardiac function. |
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Definition
1) NUCLEAR IMAGING WITH RADIOLABELED PHARMACEUTICALS, 2) CARDIAC CATHERIZATION, 3) CORONARY ANGIOGRAPHY |
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Term
100. 4 causes of impaired cardiac function. |
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Definition
1) MYOCARDIAL DISEASE, 2) VALVAR HEART DISEASE, 3) CONGENITAL HEART DEFECTS, 4) CONSTRICTIVE PERICARDITIS |
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Term
101. 4 myocardial diseases that cause impaired cardiac function. |
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Definition
1) CARDIOMYOPATHIES, 2) MYOCARDITIS PULMONARY HYPERTENSION, 3) CORONARY INSUFFICIENCY, 4) MI |
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Term
102. 2 valvar heart diseases that cause impaired cardiac function. |
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Definition
1) STENOTIC VALVULAR DISEASE, 2) REGURGITATION VULVAR DISEASE |
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Term
103. 3 causes of increased pressure work of the heart. |
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Definition
1) SYSTEMIC HYPERTENSION, 2) PULMONARY HYPERTENSION, 3) COARCTATION OF THE AORTA |
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Term
104. 2 causes of increased volume work of the heart. |
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Definition
1) ARTERIOVENOUS SHUNT, 2) EXCESS IV FLUIDS |
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Term
105. 2 causes of increased perfusion work of the heart. |
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Definition
1) THYROTOXICOSIS, 2) ANEMIA |
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Term
106. 5 etiologies of congestive cardiomyopathy. |
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Definition
1) MYOCARDIAL DAMAGE CAUSED BY DRUGS, 2) ALCOHOLISM, 3) PERIPARTUM CARDIOMYOPATHY, 4) FOLLOWING A PREVIOUS INFECTION, 5) INEFFICIENT PUMP |
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Term
107. The most common manifestations of cardiomyopathy are _____ and _____. |
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Definition
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Term
108. 5 manifestations of congestive cardiomyopathy. |
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Definition
1) DYSPNEA AND FATIGUE, 2) PALPATIONS ASSOCIATED WITH ARRHYTHMIAS, 3) CHEST PAIN, 4) ELEVATED BP, 5) EXTRA HEART SOUNDS AND CARDIAC MURMURS |
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Term
109. The prognosis of congestive cardiomyopathy is _____. |
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Definition
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Term
110. 6 treatments of congestive cardiomyopathy. |
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Definition
1) SALT RESTRICTION, 2) DIGITALIS GLYCOSIDES AND DIURETICS, 3) ANTICOAGULANTS, 4) BED REST, 5) CORTICOSTEROIDS, 6) VASODILATORS |
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Term
111. _____ is a condition caused by an increased interventricular wall septal thickness causing obstruction. |
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Definition
HYPERTROPHIC CARDIOMYOPATHY |
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Term
112. If one has hypertrophic cardiomyopathy, there will be an obstructive outcome during _____. |
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Definition
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Term
113. 3 manifestations of hypertrophic cardiomyopathy. |
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Definition
1) ANGINA, 2) DIZZINESS, 3) CHF |
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Term
114. The 2 therapies of hypertrophic cardiomyopathy is either _____ or _____. |
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Definition
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Term
115. _____ is usually caused by an infiltrative disease of the myocardium such as amyloidosis or hemochromatosis causing a stiff non-compliant heart. |
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Definition
RESTRICTIVE CARDIOMYOPATHY |
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Term
116. The most common clinical manifestation is _____. |
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Definition
RIGHT SIDED CONGESTIVE HEART FAILURE |
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Term
117. If one has restrictive cardiomyopathy, the myocardium becomes _____ and _____. |
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Definition
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Term
118. If one has restrictive cardiomyopathy, _____ is impeded and filling pressures are raised during diastole. |
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Definition
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Term
119. If one has restrictive cardiomyopathy, death occurs as a result of _____ or _____. |
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Definition
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Term
120. _____ is a group of diseases that result from the abnormal deposition of a particular protein called amyloid in various tissues of the body. |
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Definition
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Term
121. When amyloidosis occurs in a single tissue of the body is it called _____. |
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Definition
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Term
122. When amyloidosis occurs in multiple tissues in the body it is called _____, and it can cause serious changes in virtually any organ of the body. |
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Definition
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Term
123. A _____ is an out-pouching/bubbling of an artery. |
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Definition
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Term
124. A _____ aneurysm is caused by a clot. |
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Definition
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Term
125. _____ is an aneurysm when there is a tear in the aortic wall through the tunica intima and perhaps the media. |
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Definition
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Term
126. If a dissecting aneurysm results, a patient will die within _____ hours. |
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Definition
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Term
127. The most common artery affected by an aneurysm is the _____. |
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Definition
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Term
128. Abdominal aortic aneurysms most commonly occurs _____. |
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Definition
AT THE BIFURCATION OF THE ABDOMINAL AORTA INTO THE COMMON ILIAC ARTERIES |
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Term
129. The most common artery affected by a dissecting aneurysm is the _____. |
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Definition
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Term
130. 7 symptoms of a thoracic aneurysm. |
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Definition
1) SUBSTERNAL, BACK AND NECK PAIN, 2) DYSPNEA AND STRIDOR, 3) TRACHEAL PRESSURE, 4) HOARSENESS OF VOICE, 5) DIFFICULTY SWALLOWING, 6) DISTENSION OF THE NECK DUE TO SVC COMPRESSION, 7) AORTIC REGURGITATION |
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Term
131. If one has an abdominal aneurysm, a _____ may provide the first evidence of the disorder. |
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Definition
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Term
132. _____ which usually exists on the wall of an aneurysm may be detected during abdominal x-ray examinations. |
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Definition
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Term
133. If one has an abdominal aneurysm, pain may be present in the _____ and _____. |
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Definition
MID-ABDOMEN TO LOWER BACK |
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Term
134. If one has an abdominal aneurysm, the aneurysm may extend to and impinge the _____, _____, or _____ arteries. |
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Definition
RENAL, ILIAC, OR MESENTERIC |
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Term
135. If one has an abdominal aneurysm, stasis of blood favors _____ along the wall of the vessel and _____ may develop causing symptomatic arterial insufficiency. |
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Definition
THROBUS FORMATION; PERIPHERAL EMBOLI |
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Term
136. Could help rule out adjusting a patient with an abdominal aneurysm. |
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Definition
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Term
137. _____ is an acute life threatening condition that involves a hemorrhage into the vessel wall with longitudinal tearing of the vessel wall to form a blood-filled channel. |
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Definition
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Term
138. Dissecting aneurysm is caused by conditions that weaken or cause degenerative changes in the elastic and smooth muscle layer of the _____. |
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Definition
MEDIAL LAYER OF THE AORTA |
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Term
139. Dissecting aneurysms most commonly affect _____ aged _____. |
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Definition
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Term
140. 90% of dissecting aneurysm cases are associated with a previous history of _____. |
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Definition
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Term
141. The most common site of a dissecting aneurysm is the _____, while the second most common site is the _____. |
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Definition
ASCENDING AORTA; THORACIC AORTA JUST DISTAL TO THE ORIGIN OF THE SUBCLAVIAN ARTERY |
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Term
142. The major symptom of a dissecting aneurysm is an abrupt appearance of excruciating pain described as _____or _____. |
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Definition
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Term
143. Within the first 48 hours, _____% of people die who have a untreated dissecting aneurysm. |
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Definition
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Term
144. _____% of untreated dissecting aneurysm patients die within 6 weeks. |
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Definition
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Term
145. _____ is idiopathic high blood pressure that usually goes undetected due to a lack of symptoms. |
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Definition
PRIMARY/ESSENTIAL HYPERTENSION |
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Term
146. Primary/essential hypertension represents _____% of hypertension cases. |
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Definition
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Term
147. 4 causes of secondary hypertension. |
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Definition
1) CUSHINGS DISEASE, 3) PRIMARY ALDOSTERONISM, 3) PHEOCHROMOCYTOMA, 4) COARCTATION OF THE AORTA |
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Term
148. 9 risk factors of hypertension. |
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Definition
1) FAMILY HISTORY, 2) AGE, 3) MALE GENDER, 4) BLACK RACE, 5) OBESITY, 6) HIGH SODIUM DIET, 7) GLUCOSE INTOLERANCE, 8) SMOKING, 9) HEAVY ALCOHOL CONSUMPTION |
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Term
149. What is the best way to diagnose hypertension. . |
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Definition
TAKE BP AT THE SAME TIME OF DAY AND RECORD IT OVER A PERIOD OF DAYS |
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Term
150. If one has hypertension, there will be ophthalmic changes in the _____ of the eyes. |
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Definition
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Term
151. _____ is thong term effect of hypertension, so we should run kidney function tests when one presents with hypertension. |
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Definition
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Term
152. If one has hypertension, there will be a _____ in an ECG. |
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Definition
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Term
153. 7 steps in the pathyphysiology of hypertension. |
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Definition
1) HYPERTENSION DAMAGES THE WALLS OF THE SYSTEMIC VESSELS, 2) PROLONGED VASOCONSTRICTION AND HIGH PRESSURE CAUSES THE VESSELS TO THICKEN, 3) ARTERIAL SMOOTH MUSCLE UNDERGOES HYPERTROPHY, 4) TUNICA INTIMA AND MEDIA UNDERGO FIBROMUSCULAR THICKENING LEADING TO NARROWING OF THE LUMEN, 5) VESSELS BECOME PERMANENTLY NARROWED, 6) HYPERTENSIVE INJURY CAUSES INFLAMMATION TO INCREASE THE PERMEABILITY OF THE VASCULAR ENDOTHELIUM, 7) AS PERMEABILITY INCREASES MORE IONS ENTER THE VESSEL WALLS CAUSING FURTHER THICKENING |
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Term
154. When _____ occur, hypertension is usually far advanced. |
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Definition
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Term
155. 7 manifestations of hypertension. |
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Definition
1) UNSTEADINESS, 2) WALKING HEADACHE, 3) BLURRED VISION, 4) DEPRESSION, 5) RENAL DYSFUNCTION, 6) RETINAL CHANGES, 7) SIGNS OF VENTRICULAR FAILURE SUCH AS DYSPNEA OR EXERTION |
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Term
156. 4 hypertensive death related causes. |
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Definition
1) STROKE, 2) MI, 3) RENAL FAILURE, 4) ENCEPHALOPATHY |
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Term
157. _____ is a CVA when a blood clot lodges in a vessel leading to ischemic hypoxia. |
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Definition
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Term
158. _____ is a CVA when a clot comes from a far away source. |
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Definition
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Term
159. _____ is a CVA caused by bleeding. |
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Definition
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Term
160. Hypertension is the primary cause of _____ strokes. |
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Definition
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Term
161. _____ are small stroke like incidents that last 10 minutes or less. It leads to a loss in a patients senses. |
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Definition
TIA (TRANSIENT ISCHEMIC ATTACK) |
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Term
162. If one has a TIA, a patient will experience these 2 symptoms. |
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Definition
1) BRUITS, 2) THEY WILL SEE FLOATERS |
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Term
163. For something to be diagnosed as a stroke, symptoms must remain for _____ hours. |
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Definition
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Term
164. If a patient has a thrombotic stroke, _____ can be administered to bust out the clot that’s blocking the brain, and restore blood flow. This is able to completely resolve the stroke leading too complications. |
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Definition
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Term
165. Strokes tame time to _____ to completion. It might not look severe at first, but the next day the patient could look much worse. |
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Definition
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Term
166. Once a patient has completed a stoke they should work on _____ as quick as possible. |
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Definition
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Term
167. _____ and _____ are 2 agents used to treat thrombotic and embolic strokes. They must not be administered to a patient with a hemorrhagic stroke because they will bleed to death. |
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Definition
FIBRINOLYSIS AND FIBRINOLYTIC AGENTS |
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Term
168. 13 risk factors of cerebrovascular disease. |
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Definition
1) ATHEROSCLEROSIS, 2) HYPERTENSION, 3) HIGH CHOLESTEROL, 4) FAMILY HISTORY, 5) OBESITY, 6) SEDENTARY LIFESTYLE, 7) ORAL CONTRACEPTIVES, 8) SMOKING, 9) COAGULATION DISORDERS, 10) DEHYDRATION, 11) CHRONIC HYPOXIA, 12) CIGARETTE SMOKING |
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Term
169. 4 manifestations of a left hemispheric stroke. |
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Definition
1) RIGHT SIDED HEMIPLEGIA, 2) SLOW AND CAUTIOUS BEHAVIOR, 3) RIGHT VISUAL FIELD DEFECT, 4) EXPRESSIVE, RECEPTIVE OR GLOBAL DYSPHASIA |
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Term
170. 7 manifestations of a right hemispheric stroke. |
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Definition
1) LEFT SIDED HEMIPLEGIA, 2) SPATIAL-PERCEPTUAL DEFICITS, 3) IMPULSIVE BEHAVIOR, 4) DISTRACTABILITY, 5) DENIAL, 6) LEFT VISUAL FIELD DEFECTS, 7) PERSEVERATION (REPEATING A WORD OVER AND OVER) |
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