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*scala naturae *discrete, unchanging (essentialism) *everything derived from study of geometry *variation provides raw material for evolution |
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Geologists said earth was older by |
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*gradual sedimentation produced thick rock layers *gradual erosion, produced deep valleys |
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Jean Baptiste de Lamarck theories |
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*fossils linked through different sedimentary deposits leading to now forms *evolution gradual, long time *changing environment= changing evolution *reject essentialism *evolution slow *evolution occurs because strive for perfection |
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Evolution by NS produces complexity because |
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*blind process produces adaptations which accumulates over long time |
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*religious chain of beings rather than adaptation |
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*showed the world was changing *deeper strata showed more extinct animals *shallow strata was more modern animals *fish-reptiles-birds: shallower sedimentary |
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Darwin and Wallace Theories |
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*evolution occurs as automatic process that produces adaptation *Lamarack was incorrect spontaneous generation *Lamarck correct about long time evolution |
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*organisms derived from single first appearance |
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*rejected blending *no inheritance= no evolution |
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*Inheritance blending or particulate *accepted particulate *A1A2 heterozygotes produce two types of gametes, one carrying the A1 allele and the other carrying the A2 allele |
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*Independent Assortment *different chromosomes move independent *genetic recombination |
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*meiosis + fertilization, only homologous *crossing over |
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*reside close on same chromosome *autozomal *does not satify 2nd law |
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Darwin proposed NS but did not understand |
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* if inheritance was blending, NS would be ineffective in producing evolutionary change *if inheritance occurred through a particulate mechanism, NS would be effective in producing evolutionary change |
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*during DNA replication *most important source of genetic variation *inherited by germ cells |
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Assume: 1) Lrg Population 2) Random mating 3) No gene flow 4) No on-going mutation 5) No selection (no diff in repro success) |
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Change heterozygote as frquency A allele changes |
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Move from relative freq to absolute frequency |
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divide by the sum of the relative frequencies |
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Selection against dominant allele |
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*rapidly drive the allele frequency to zero, because selection is so strong (s = 1) *produce equilibrium genotype frequencies that are predicted by the Hardy-Weinberg equilibrium *generate increased genetic variability in the population, much like mutation * produce the most rapid changes in allele frequencies when the initial frequency of the recessive allele is low |
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*population bottleneck *founder effect |
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*pop becomes small quickly *catastrophic event *alleles lost, genetic variation dec |
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*colonize new location speaparte from original *reduced pop size, red genetic vary |
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Genetic drfit: small population |
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greater effect b/c gene pool small |
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Influences of genetic drift |
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*population bottleneck after catastrophe *low pop size *founder event |
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Genetic Drift: both pheno on separate islands NS: one phenotype more favored |
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*no change in mean * increase in variance *allows Random mating to produce low-fitness intermediate phenotypes *fitness values are highest for individuals whose trait values are closest to the mean |
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*no change in mean *decrease in variance |
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*change in mean *little change in varince *ind @ one end has more fitness than ind @ other end |
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Frequency distributions of phenotypic traits |
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*bell shaped curve *environmental effects produce variation in trait value |
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predators evovle resistance slower than prey |
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*predators that survive exposure to an insecticide may have a low fitness, because their insect prey population may be eliminated by the insecticide |
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*produces a protein that is toxic to some insects but not to vertebrates |
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*non transgenic *represent a source population that can produce susceptible individuals that migrate into transgenic field |
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*greater per capita competitive effect with other species *inter *same food, diff quant |
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*same size *eat different things *intra |
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*conversion efficiency from prey-pred *the number of predator births occurring for each prey consumed |
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pred per capita death rate |
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Prey lost from population |
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1) death 2) emigration 3) eaten by predator |
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[image] density dependent positive feedback not good regulator |
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Selection against a recessive lethal allele weakens as the frequency of the recessive allele approaches zero because |
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c. selection can only act on the lethal allele when it is in a homozygote, and homozygous individuals become very rare as the recessive allele’s frequency drops |
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*detoxification, target site initiated *detox fail *stable source of immigrating crop pests that are nearly all genotype SS *any RR individuals that survive exposure to the toxin being likely to mate with SS individuals, producing RS offspring *initial frequency of the R allele be low enough that RR individuals are not common *resistance evolution is slowed by expressing a toxin dose that is sufficiently high to kill the rare individuals that are heterozygous for the resistance-conferring allele (RS individuals) |
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SS- susceptible RS- resistant RR-resistant |
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SS- susceptible RS- susceptible RR- resistant |
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Selection against a deleterious recessive allele |
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*acts against homozygotes that carry two copies of the deleterious recessive allele *“slow down” as the frequency of the allele gets very small *when the allele is rare in natural selection |
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*can contribute to changes in allele frequencies, even when those same alleles are influenced by natural selection |
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Population dynamics of predators and their prey |
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*often involve negative feedback cycles, but with a time-lag, resulting in cycles of prey and predator populations over time. |
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Reciprocal adaptations in herbivores and plants |
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exhibit a stepwise coevolutionary process of adaptation and counter-adaptation |
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predatory insects rarely evolve resistance |
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*herbivorous insects that survive an insecticide application are expected to have high fitness *predatory insects that survive an insecticide application may still have a low fitness |
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*can evolve when the ‘model’ species is defended (e.g., by toxic chemicals), but the ‘mimic’ is not *can evolve when multiple defended species evolve to resemble each other |
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Zero growth isoclines for interspecific competition and predator-prey interactions |
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*are lines along which that population’s growth rate is expected to be zero *separate an x-y plot into two regions, one in which the population is expected to grow and the other in which the population is expected to shrink |
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*can cause different genes residing very far apart on the same chromosome to exhibit independent assortment, or at least approach the condition of independent assortment |
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* strong immigration of susceptible individuals from a nearby crop field that does not produce the protein toxin *a dose of the toxin that is high enough to kill individuals with genotype RS (i.e., heterozygotes carrying one resistance-conferring allele and one susceptibility-conferring allele) *matings between rare RR individuals and SS individuals, producing RS offspring *a dose of the toxin that is high enough to kill RS individuals and most of the rare RR individuals |
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*the population has a single stable equilibrium where the birth rate and the emigration rates are equal |
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Violate pattern of mendelian inheritance |
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*when two genes are found close together on the same autosome (autosomal linkage) *when genes of interest are found on sex chromosomes |
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Insecticide resistance is less likely to evolve in populations of predatory insects |
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*predators that survive an insecticide application may still have low fitness, because they may have no prey to eat |
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