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| Corynebacterium diptheriae |
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| Mycobacterium tuberculosis |
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| opportunistic/nosocomial infections |
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| Inflammatory disease of large intestine |
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| Enterohemorrhagic E. Coli |
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| hemorrhagic syndrome and kidney damage |
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| bubonic; septicemic; pneumonic |
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O2 PREFERENCES:
Bacillus anthracis |
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O2 PREFERENCES:
Clostridium perfringens |
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O2 PREFERENCES:
Clostridium tetani |
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O2 PREFERENCES:
Clostridium difficile |
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O2 PREFERENCES:
Clostridium botulism |
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O2 PREFERENCES:
Listeria monocytogenes |
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O2 PREFERENCES:
Corynebacterium diptheriae |
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O2 PREFERENCES:
Mycobacterium tuberculosis |
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O2 PREFERENCES:
Mycobacterium leprae |
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O2 PREFERENCES:
Pseudomonas aeruginosa |
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O2 PREFERENCES:
Francisella tulerensis |
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O2 PREFERENCES:
Bordetella pertussis |
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O2 PREFERENCES:
Legionella pneumophila |
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O2 PREFERENCES:
GENERAL E.COLI |
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O2 PREFERENCES:
Salmonella typhii |
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O2 PREFERENCES:
Salmonella enteritidis |
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O2 PREFERENCES:
Yersinia pestis |
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O2 PREFERENCES:
Haemophilus influenzae |
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O2 PREFERENCES:
Haemophilus aegyptius |
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-cell-mediated immunity against virus-infected cells and cancer cels
-produced in bone marrow, mature in thymus |
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| How are T-Cells activated? |
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Definition
1) A phagocyte eats a bacteria
2) parts of the bacteria (antigen) goes to the surface of the phagocyte
3) The phagocyte presents the antigen to a helper T cell |
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| Antigen presents on class II is from the _______. |
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| Function of Helper T-Cell |
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-helps B cells become active
-secrete cytokines that help many immune cells function/become activated |
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Cytotoxic T-Cells Function
(KILLER T CELLS) |
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-will secrete a toxin (granzyme & perforin "pokes holes in cell membrane of infected cell")->causes cell to lose integrity
-Granzyme is secreted in to kill the cell |
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-coordinated/programmed cell death
-no bystanders |
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-the ability to combat diseases and cancer
-can be brough about naturally through an infection or artificially through medical intervention.
-two types active/passive |
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-the indiv. body makes antibodies against a particular antigen
-primary exposure is shorter lived and slower to respond while a secondary exposure is a rapid, strong response long lasting
-depends on memory B and T cells |
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-an indiv. is given prepared antibodies against a particular antigen
-short lived immunity
-mother to child immunity passage |
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| -after first exposure to an Ag immune system produces antibodies that gradually increase |
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| -after second contact with the same Ag, immune system produces a more rapid, stronger response due to memory cells |
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| Compare and contrast capsules, surface proteins, and exotoxins. |
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