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"small ear" congenital deformity of pinna occurs in 1 out of 8000-10,000 births treatment- surgery 3 groups: |
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AKA hematoma auris condition among boxers external portion suffers a bow, a blood clot formation of fibrous tissue in overlying skin may causes CHL |
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"no hole", closed off ear canal unilateral is 6 times more common than bi CHL treatment- surgery to make canal |
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benign bony growths only significant if they occlude entire canal caused by extended exposure to cold wind/water "surfer's ear" HL- minimal no treatment |
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Swimmer's Ear inflammation of EAC swelling, pain, drainage, smell caused-common bacteria CHL treatment- meds |
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hole in TM CHL opens up ME to intrusion heals on their own treatment- myringoplasty |
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white plaques form on TM changes in connective tissue layer abnormal deposits of calcium no HL no treatment |
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Eustachian Tube dysfunction causes |
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inflammatory tissue change in nasopharynx physical diff in anatomy growth of mass in the nasopharynx neg pressure created when ET is blocked CHL |
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abnormal growth behind ear drum in ME cysts or puches that shed layers of old skin increase in size and destroy surrounding bones in ME CHL treatment-removal |
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embryonal epithelial cells white mass medial to normal TM |
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ET dysfunction Poor aeration of the epitympanic space retraction of pars flaccida keratin accumulates, sac is enlarged |
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exudate- fluids leak from membranes transudate- neg pressure pulls fluid from cells purulent- fluid becomes infected |
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ossicles fuse together-immovable mass progressive disorder HL-bilateral, Carhart Notch may be hereditary white, middle-aged women most at risk treatment- nothing, HAs, sugery |
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temporal bone fracture traumatic membrane perforation TM may or may not be perforated HL treatment- prosthetic ossicle |
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abnormality of IE bilateral "episodes" HL- low freq SNHL vertigo, tinnitus, pressure/pain no cure |
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affects stereocilia of hair cells overstimulated ear aka acoustic trauma SNHL- typically 4000 Hz no treatment to restore hearing TTS and PTS |
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age-related HL progressive, bilateral SNHL- higher freq's no treatment, but HAs |
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meds cause HL balance dysfunction/tinnitus SNHL HAs, stop meds chemo drugs, mycin drugs, loop diurectics, asprin |
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develops instantly/instantaneously HL- unilateral often waking up in morning call lose all hearing/can be mild viral infection, vascular insult, IE rupture |
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Autoimmune Inner Ear Disease (AIED) |
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immune system causes body to attack its own tissues attacks IE HL- bilateral, progressive SNHL treatment- steroids |
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sudden changes in pressure rupture of OV or RW fistula SNHL treatment- patch the leak |
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Benign Paroxysmal Positional Vertigo |
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small crystals come loose and float around in IE cause vertigo audiogram and vestib tests certain positions short duration |
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neuromass- mass on 8th nerve benign tissue diagnosed via MRI remove tumor, often patients lose hearing in that ear |
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inherited/genetically transmitted multiple neuromas on sensory nerves bilateral HL treatment- HAs, remove neuromas |
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inflammation/infection in middle ear -HL: in most cases Tx: tubes or antibiotics if infected |
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WITHOUT EFFUSION: inflammation no fluid WITH EFFUSION: fluid without S&S infection 'serous otitis' 'glue ear' ACTUE OM: fluid rapid onset s&s infection CHRONIC OM: infection persists |
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OM with Effusion sometimes called |
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glue ear Serous Otitis Media SOM |
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ongoing dx to middle ear and eardrum patient will have language disorder |
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Exudate Transudate Purulent |
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fluid leaks from membranes due to inflammation in ME space |
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(-) pressure pulls fluid from cells in lining of ME |
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resulting fluid becomes infected, containing pus |
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Most common Dx in sick children in US |
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-respiratory infection can lead to OM -daycare -bottle feeding -fever ear pain, fussiness, feeding problems |
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cleft palat, downs, allergies -native americans/eskimos higher risk -under age 5 who have their first ear infection before 6 mo. |
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ossicles become fused together into immovable mass formation fo spongy bone over stepdial footplate |
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bilateral, Carhart Notch (2000 Hz sensorineural) |
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Otosclerosis is a ____ Disorder |
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-do nothing -HA for CHL -Stapdectomy |
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temporal bone fracture Traumatic Tympanic Membrane Perforation (may/may not) Ossicles disengaged and not in chain |
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-Meniere -NIHL -Presbycusis -Ototoxicity -Sudden SNHL -AIED -Barotrauma |
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IE DISORDER 'episodes' fluid & fluid retnetion in inner ear HL: low frequency SNHL (200-8000Hz) *** Tx: diet medications for vertigo endolymphatic shunt |
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Hearing Loss from Noise Exposure |
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Mechanical: affects steriocilia Biochemical: overstimulated ear can't maintain appropriate chemical balances |
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NIHL has HL: TX: Noise Exposure Terms: |
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HL: SNHL, 4000 Hz Notch Tx: no treatment -- EDUCATE PEOPOLE Terms: TTS - Temporary Threshold Shift PTS: Permanent Threshold Shift OSHA: Occupational Safety and Health Administration |
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age-related hearing loss -men early 60s women late 60s progressive bilateral symmetrical SNHL -sensory cells fo inner ear cannot repair -ypically HL at Higher Requency TX: no medical tx but HA for SNHL |
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Capacity for certain medications to cause hearing loss: |
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-Antineopastic -chemo Aminoglycoside Antibiotics: mycin Loop Diuretic: fluid Analgeics: asprin |
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Ototoxicity associated with |
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balance dysfunction tinnitus SNHL, may resolve when stop taking meds |
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develops instantaneously unilateral HL ISSHL -wake up in morning |
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3 Causes of Sudden Onset SNHL |
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1. viral 2. vascular insult 3. fistula (inner ear rupture) |
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inflammatory conditions that occur when immune system causes body to attack it's own tissues: |
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AIED: AutoImmune Inner Ear Disease |
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bilateral, fluctuating progressive SNHL |
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sudden chagnes in pressure rupture of round/oval window Fistula (perillymph leak) can patch fistula |
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Crystals lose and float inside inner ear: |
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BPPV: Benign Paroxysmal Positional Vertigo |
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Rupture of Oval or Round Window may cause |
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Incidence of Acoustic Tumors/Acoustic Neuromas |
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